DEFINITION
: this is an acute pneumonia involving one or more lobes of the lung characterized by sudden onset, chill, fever, difficulty in breathing, cough, and blood-stained sputum, marked by consolidation, and normally followed by resolution and return to normal of the lung tissue
STAGES
Lobar pneumonia usually has an acute progression. Classically, the disease has four stages:
Congestion in the first 24 hours: This stage is characterized histologically by vascular engorgement, intra-alveolar fluid, small numbers of neutrophils, often numerous bacteria. Grossly, the lung is heavy and hyperemic
Red hepatization or consolidation : Vascular congestion persists, with extravasation of red cells into alveolar spaces, along with increased numbers of neutrophils and fibrin. The filling of airspaces by the exudate leads to a gross appearance of solidification, or consolidation, of the alveolar parenchyma. This appearance has been likened to that of the liver, hence the term "hepatization".
Grey hepatization : Red cells disintegrate, with persistence of the neutrophils and fibrin. The alveoli still appear consolidated, but grossly the color is paler and the cut surface is drier.
Resolution (complete recovery):The exudate is digested by enzymatic activity, and cleared by macrophages or by cough
HOW IT IS DIAGNOSED
The most common organisms which cause lobar pneumonia are Streptococcus pneumoniae, also called pneumococcus, Haemophilus influenzae and Moraxella catarrhalis.
Mycobacterium tuberculosis, the tubercle bacillus, may also cause lobar pneumonia if pulmonary tuberculosis is not treated promptly.
The identification of the infectious organism (or other cause) is an important part of modern treatment of pneumonia. The anatomical patterns of distribution can be associated with certain organisms,and can help in selection of an antibiotic while waiting for the pathogen to be cultured.
TREATMENT
See treatment for pneumonia. http://www.talktonurserita.com/search?q=pneumonia&x=5&y=8
: this is an acute pneumonia involving one or more lobes of the lung characterized by sudden onset, chill, fever, difficulty in breathing, cough, and blood-stained sputum, marked by consolidation, and normally followed by resolution and return to normal of the lung tissue
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| X-RAY RESULTS |
STAGES
Lobar pneumonia usually has an acute progression. Classically, the disease has four stages:
Congestion in the first 24 hours: This stage is characterized histologically by vascular engorgement, intra-alveolar fluid, small numbers of neutrophils, often numerous bacteria. Grossly, the lung is heavy and hyperemic
Red hepatization or consolidation : Vascular congestion persists, with extravasation of red cells into alveolar spaces, along with increased numbers of neutrophils and fibrin. The filling of airspaces by the exudate leads to a gross appearance of solidification, or consolidation, of the alveolar parenchyma. This appearance has been likened to that of the liver, hence the term "hepatization".
Grey hepatization : Red cells disintegrate, with persistence of the neutrophils and fibrin. The alveoli still appear consolidated, but grossly the color is paler and the cut surface is drier.
Resolution (complete recovery):The exudate is digested by enzymatic activity, and cleared by macrophages or by cough
HOW IT IS DIAGNOSED
The most common organisms which cause lobar pneumonia are Streptococcus pneumoniae, also called pneumococcus, Haemophilus influenzae and Moraxella catarrhalis.
Mycobacterium tuberculosis, the tubercle bacillus, may also cause lobar pneumonia if pulmonary tuberculosis is not treated promptly.
The identification of the infectious organism (or other cause) is an important part of modern treatment of pneumonia. The anatomical patterns of distribution can be associated with certain organisms,and can help in selection of an antibiotic while waiting for the pathogen to be cultured.
TREATMENT
See treatment for pneumonia. http://www.talktonurserita.com/search?q=pneumonia&x=5&y=8
